基本信息·出版社：北京大学医学出版社 ·页码：472 页 ·出版日期：2009年08月 ·ISBN：7811164868/9787811164862 ·条形码：9787811164862 ·版本：第1 ...
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病理学(第8版)(英文改编版) |
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基本信息·出版社：北京大学医学出版社
·页码：472 页
·出版日期：2009年08月
·ISBN：7811164868/9787811164862
·条形码：9787811164862
·版本：第1版
·装帧：平装
·开本：16
·正文语种：中文/英语
·丛书名：双语教材，国外经典医学教材改编/影印系列
·外文书名：Textbook of Pathology
内容简介 《病理学(第8版)(英文改编版)》依据Robbins Basic Pathology的最新版第8版进行改编。在改编过程中，对《病理学(第8版)(英文改编版)》的内容进行了删节、调整和适当补充，个别章节有较大的更新和改动。同时，在内容上兼顾了临床医学及其他相关专业和不同学制的需求。因此《病理学(第8版)(英文改编版)》可用作临床医学、口腔、公共卫生专业的五年制、七年制、八年制和留学生的双语教学用书，也可作为病理医生和进修生的重要参考书，以及作为执业医师资格考试的复习用书。
编辑推荐 《病理学(第8版)(英文改编版)》是专门为中国医学生打造的病理学双语教材。由国内20家医学院校的病理学教授联合美国华裔病理学者依据Robbins Basic Pathology的最新版（第8版）进行改编。改编以国内教学大纲为依据，对原版内容进行了删节、调整和适当补充。改编版教材既保留了原版精华，保证教材权威性，又能够最大限度地适应国内双语教学需求。
供医学各专业本科生、留学生、长学制、研究生双语教学使用。
目录 Introduction to Pathology
What is Pathology?
How to Study Pathology?
Diagnostic and Research Techniques in Pathology
What Do Pathologists Do?
The History and Evolution of Pathology
Pathology in China
Chapter 1 Cell Injury, Cell Death, and Adaptations
Cellular Adaptations to Stress
Cell Injury and Cell Death
Cellular Aging
Chapter 2 Tissue Repair: Regeneration, Healing, and Fibrosis
The Regeneration
The Nature and Mechanisms of Action of Growth Factors
Extracellular Matrix and Cell-Matrix Interactions
Cell and Tissue Regeneration
Repair by Connective Tissue
Cutaneous Wound Healing
Fracture HealingPathologic Aspects of Repair
Chapter 3 Hemodynamic Disorders
Edema
Hyperemia and Congestion
Hemorrhage
Hemostasis and Thrombosis
Embolism
Infarction
Chapter 4 Acute and Chronic Inflammation
Overview of Inflammation
Acute Inflammation
Chronic Inflammation
The Possible Outcomes of Inflammation
The Significance of the Inflammatory Response
Chapter 5 Diseases of the Immune System
Autoimmune Diseases
Rejection of Transplants
Immune Deficiency Diseases
Chapter 6 Neoplasia
Definition and General Morphology
Differentiation and Anaplasia of Tumors
Growth and Spread of Tumors
Effects of Tumor on Host
Identification Between Benign and Malignant Tumors
Nomenclature and Classification of Tumors
Brief Introduction of Common Neoplasms
Carcinogenesis and Etiology of Neoplasia
Host Defense Against Tumors: Tumor Immunity
Chapter 7 Genetic, Environmental and Nutritional Diseases
Genetic Diseases
Mendelian Disorders (Diseases Caused by Single-Gene Defects)
Disorders with Multifactorial Inheritance
Cytogenetic Disorders
Environmental Diseases
Environmental Pollution and Occupational Exposures
Addictions and Associated Diseases
Injury by Physical Agents
Nutritional Diseases
Malnutrition
Protein-Energy Malnutrition (PEM)
Vitamin Deficiencies
Obesity
Chapter 8 Cardiovascular System
The Heart
Ischemic Heart Disease (IHD)
Hypertensive Heart Disease
Valvular Heart Disease
Cardiomyopathies
Pericardial Disease
The Blood Vessels
Arteriosclerosis
Atherosclerosis
Hypertensive Vascular Disease
Chapter 9 The Lung
Pulmonary Infections
Obstructive Pulmonary Disease
Diseases of Vascular Origin
Diffuse Interstitial (Restrictive, Infiltrative) Lung Diseases
Tumors of Respiratory System
Chapter 10 The Gastrointestinal Tract
Esophagus
Esophagitis
Barrett Esophagus
Esophageal Carcinoma
Stomach
Gastritis
Gastric Ulceration
Gastric Tumors
Small and Large Intestines
Inflammatory Bowel Disease
Tumors of the Small and Large Intestines
Appendix
Acute Appendicitis
Tumors of the Appendix
Chapter 11 The Liver, Gallbladder, Biliary Tract, and Pancreas
The Liver
Pattems of Hepatic Injury
Viral Hepatitis
Autoimmune Hepatitis
Drug-induced Liver Disease
Alcoholic Liver Disease
Metabolic Liver Disease
Cirrhosis
Malignant Tumors of The Liver Origin
Disorders of the Gallbladder
Cholelithiasis (Gallstones)
Cholecystitis
Carcinoma of The Gallbladder
The Pancreas
Pancreatitis
Pancreatic Carcinoma
Chapter 12 The Hematopoietic and Lymphoid Systems
White Cell Disorders
Non-Neoplastic Disorders of White Cells
Neoplastic Proliferations of White Cells
Chapter 13 The Kidney and Its Collecting System
Clinical Manifestations of Renal Diseases
Glomerular Diseases
Diseases Affecting Tubules and Interstitium
Tumors
Chapter 14 The Male Genital System
Penis
Scrotum, Testis, and Epididymis
Prostate
Chapter 15 The Female Genital System and Breast
Cervix
Cervicitis
Tumors of the Cervix
Body of Uterus
Endometriosis
Endometrial Hyperplasia
Cbapter 16 The Endocrine System
Cbapter 17 The Musculoskeletal System
Chapter 18 The Nervous System
Chapter 19 Pathology of Infectious Diseases
Chapter 20 Parasitosis
……
序言 为使医学教育和国际逐步接轨，双语教学在我国医学院校已推行多年，但至今仍然缺乏被广泛认可的教科书。自编的教材受英语水平的限制，语言表达上往往不尽人意。
Robbius Basic Pathologp，是在全世界深受欢迎并被广泛采用的权威性教学用书，其立足前沿的理论知识、独特精致的编写风格、严谨规范的专业术语、图文交融的编排方式，无一不受到广大医学生和病理学工作者的推崇和青睐。是美国医学生学习病理学的首选教材，病理医师资格考试的必读用书，也是我国中文病理教材编写的主要参考书。
Robbins Basic Pathologpr原版的国际版和影印版已在国内销售，但由于价格依然偏高，某些内容与中国的教学习惯不完全吻合，尚难作为病理学教科书广泛应用。基于该书在国际上的影响力及我国的医学教育现状，北京大学医学出版社决定与ELSEVIER公司联合对该书进行改编。其目的是在不改变原书风格和基本内容的前提下，通过改编、精编和缩编使其内容和编排顺序符合中国的教学习惯。贴近前沿、贴近临床、贴近我国的教学实际是本书改编的主要宗旨。
本书依据Robbins Basic Pathology的最新版第8版进行改编。在改编过程中，对本书的内容进行了删节、调整和适当补充，个别章节有较大的更新和改动。同时，在内容上兼顾了临床医学及其他相关专业和不同学制的需求。因此本书可用作临床医学、口腔、公共卫生专业的五年制、七年制、八年制和留学生的双语教学用书，也可作为病理医生和进修生的重要参考书，以及作为执业医师资格考试的复习用书。
本书编委均来自教学第一线，在双语教学和教材编写方面均具有丰富的经验。在繁重的病理教学、科研和诊断工作的同时，大家辛勤劳作，不遗佘力地完成了初稿的编写；另有几位美国的病理学同道也参与了本书的编写。最后，又经美国的病理学专家翟启辉教授修改润色，反复斟酌，力求行文准确、简明易懂，体现原书的学术水平和语言风格。本书的编委会秘书陈方杰女士和山东大学病理学教研室的吴晓娟医师做了大量卓有成效的工作，山东大学病理学教研室的李丽、吴晓娟、项磊、张晓芳和桂婷参与了部分章节的二校，出版社的责任编辑也付出了辛勤劳动，在此一并表示感谢。另外，本书原版主编Kumar教授对本书的改编给予热情支持，并在百忙之中欣然作序，我们在享受其学术成果的同时，在此谨致由衷的谢意。
改编是双语教材编写的尝试和探索，疏漏和错误在所难免，愿广大病理学同道和学生在使用中不断提出宝贵意见，以期再版时不断完善。
文摘 插图：
Intracellular Accumulations
Under some circumstances cells may accumulate abnormalamounts of various substances, which may be harmless orassociated with varying degrees of injury. The substancemay be located in the cytoplasm, within organelles（typically lysosomes）, or in the nucleus, and it may besynthesized by the affected cells or may be producedelsewhere.
There are three main pathways of abnormal intracellularaccumulations:
A normal substance is produced at a normal or anincreased rate, but the metabolic rate is inadequate toremove it. An example of this type of process is fattychange in the liver.
A normal or an abnormal endogenous substanceaccumulates because of genetic or acquired defects inits folding, packaging, transport, or secretion. Mutationsthat cause defective folding and transport may lead to accumulation of proteins （e.g., l-antitrypsin deficiency）.
An abnormal exogenous substance is deposited andaccumulates because the cell has neither the enzymaticmachinery to degrade the substance nor the ability totransport it to other sites. Accumulations of carbon or silica particles are examples of this type of alteration.Fatty Change （Steatosis）. Fatty change refers to anyabnormal accumulation of triglycerides within paren-chymal cells. It is most often seen in the liver, since this isthe major organ involved in fat metabolism, but it may alsooccur in heart, skeletal muscle, kidney, and other organs.
Hepatic steatosis may be caused by toxins, proteinmalnutrition, diabetes mellitus, obesity, and anoxia.Alcohol abuse and diabetes associated with obesity arethe most common causes of fatty change in the liver （fattyliver） in industrialized nations.
Free fatty acids from adipose tissue or ingested foodare normally transported into hepatocytes. Excessaccumulation of triglycerides may result from defectsat any step fro
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